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Dive into the research topics where Marc Tambini is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Collaborations and top research areas from the last five years
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Presenilin Knock-in Rat Model of Neurodegeneration
Tambini, M. (PI)
2/10/23 → 1/31/26
Project: Research
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Presenilin Knock-in Rat Model of Neurodegeneration
Tambini, M. (PI)
5/15/21 → 4/30/23
Project: Research
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Analysis of early effects of human APOE isoforms on Alzheimer’s disease and type III hyperlipoproteinemia pathways using knock-in rat models with humanized APP and APOE
Yesiltepe, M., Yin, T., Tambini, M., Bao, H., Pan, M., d’Abramo, C., Giliberto, L., Han, X. & Luciano, D., Sep 2024, In: Cell Communication and Signaling. 22, 1, 458.Research output: Contribution to journal › Article › peer-review
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Correction to: Analysis of early effects of human APOE isoforms on Alzheimer’s disease and type III hyperlipoproteinemia pathways using knock-in rat models with humanized APP and APOE (Cell Communication and Signaling, (2024), 22, 1, (458), 10.1186/s12964-024-01832-2)
Yesiltepe, M., Yin, T., Tambini, M., Bao, H., Pan, M., d’Abramo, C., Giliberto, L., Han, X. & D’Adamio, L., Dec 2024, In: Cell Communication and Signaling. 22, 1, 619.Research output: Contribution to journal › Comment/debate › peer-review
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Aβ43 levels determine the onset of pathological amyloid deposition
Tambini, M. D., Yin, T., Yesiltepe, M., Breuillaud, L., Zehntner, S. P., d'Abramo, C., Giliberto, L. & D'Adamio, L., Jul 2023, In: Journal of Biological Chemistry. 299, 7, 104868.Research output: Contribution to journal › Article › peer-review
Open Access -
Late-long-term potentiation magnitude, but not Aβ levels and amyloid pathology, is associated with behavioral performance in a rat knock-in model of Alzheimer disease
Yesiltepe, M., Yin, T., Tambini, M. D., Breuillaud, L., Zehntner, S. P. & D’Adamio, L., Nov 11 2022, In: Frontiers in Aging Neuroscience. 14, 1040576.Research output: Contribution to journal › Article › peer-review
Open Access -
Knock-in rats with homozygous PSEN1L435F Alzheimer mutation are viable and show selective γ-secretase activity loss causing low Aβ40/42 and high Aβ43
Tambini, M. D. & D'Adamio, L., May 22 2020, In: Journal of Biological Chemistry. 295, 21, p. 7442-7451 10 p.Research output: Contribution to journal › Article › peer-review
Open Access5 Scopus citations